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Glucagon receptor agonism drives significant increases in energy expenditure and hepatic fat oxidation effects that are largely independent of the insulin secretion and appetite pathways activated by GLP-1R and GIPR

These trials will provide definitive efficacy and safety data needed for regulatory submissions, with results expected in the coming years

Because that term is in a lot of people's consciousness. GLP-1 drugs mimic a hormone the body produces after eating, which can decrease appetite

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