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Although this difference approached but did not reach the threshold for clinical significance established in other studies, 134 the findings suggest the potential neuroprotective effects of lixisenatide, possibly mediated by increased synaptic dopamine levels, as supported by preclinical and clinical research in addictive disorders

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(Santa Cruz, CA, United States) and against Heme Oxygenase-1(HO-1), PPAR- SIRT-1, Glut-4, and Aconitase from Abcam (Cambridge, United Kingdom) and for Hsp-70 from Sigma-Aldrich Fine Chemicals (St Louis, MO, United States)