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Intracellular ROS homeostasis is characterized by the ability of cells to adapt to changing conditions such as hypoxia, hyperoxia, and oxidative stress [4,5,6], while disruptions in this equilibrium can have significant implications for cell fate [7], disease progression [8], and therapeutic responses [9]

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1 Introduction Fibrosis is characterized by the destruction of fibrous connective tissue, activation and proliferation of fibroblasts, elevated collagen fiber secretion, and excessive deposition of extracellular matrix (ECM) (Zhang and Zhang, 2020)

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