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Pathogenic VCP mutations induce mitochondrial uncoupling and reduced ATP levels

there is the potential for: Ischemic injury due to potent vasoconstrictor action and tissue hypoxia Mild bradycardia, probably as a reflex result of a rise in blood pressure Arrhythmias Anxiety Transient headache Respiratory difficulty Pharmacokinetics: When released from nerve terminals, its actions are primarily (75%) terminated by neuronal reuptake Diffusion and degredation by extracellualr COMT contribute to a lesser extent

Andrade G, Gorgulho B, Lotufo P, Bensenor I, Marchioni D (2018) Dietary selenium intake and subclinical hypothyroidism: a cross-sectional analysis of the ELSA-Brasil study

In the rabbit bone defect study, BPC-157 treatment produced complete bony continuity across the defect site within six weeks in treated animals, while all control animals remained unhealed

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