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Inside the mitochondria, carnitine and acyl-CoA are then regenerated from the internalized acylcarnitines by CPTII before the acyl-CoA is catabolized in two-carbons units by oxidation by the mechanism described below with generation of acetyl-CoA as the main product in normal circumstances

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SNAP/FLAG-hGLP-1R degradation is quantified as the inverse of total residual receptor signal normalised to non-stimulated wells after subtracting non-labelled background signal

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