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At the neurochemical level, preclinical studies demonstrate that GLP-1 RAs modulate dopamine levels and glutamatergic neurotransmission in reward circuits, attenuate accumbal dopamine release in response to drugs of abuse and suppress phasic dopamine responses to food-predictive cues in the VTA

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Tirzepatide demonstrated that adding GIP activation to GLP-1 agonism produced meaningfully better outcomes than GLP-1 alone