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It has been reported that Dihydroquercetin (DHQ) inhibited ferritin autophagy and decreased ferrous iron in unstable intracellular iron pools by downregulating microtubule-associated protein 1 A/1B-light chain 3 (LC3) and upregulating ferritin heavy chain 1 (FTH1), as well as nuclear receptor co-activator 4 (NCOA4), in activated HBE cells for silicosis treatment [27]

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As shown in Fig.6h, D-CuP exhibited significantly stronger binding affinities, with equilibrium dissociation constants (KD) of 3.8 10 M for p53 and 1.81 10 M for HDAC7

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This mechanism is supported by Pickarts foundational research on GHK-Cu and tissue remodeling, which established the peptides role in accelerating wound closure and improving the structural quality of regenerated tissue