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Furthermore, NaB-enhanced GLP-1R expression in HepG2 cells by inhibiting histone deacetylase-2 independent of GPR43/GPR109a

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The ILK/PI3K/Akt survival signalling activated by TB-500 is also relevant to neuronal survival following ischaemic and traumatic CNS injury , providing an anti-apoptotic neuroprotective mechanism alongside the pro-migratory effects on neural repair cell populations

This article is not an argument against their use

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