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SHANK3 mutations identified in autism lead to modification of dendritic spine morphology via an actin-dependent mechanism

In cellular and animal models, Cagrilintide exposure has also been associated with measurable changes in metabolic biomarkers, alterations in feeding related signaling circuits, and modulation of pathways connected to gastric motility and energy balance [6]

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The in vitro hippocampal-neuron-culture spine-density work used Dihexa concentrations across a wide range, with effects observed at picomolar concentrations [2]