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Lowered inflammation and re-balanced hormones result in noticeable fertility improvements

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Our data revealed the broad influence of GSTM1 on the regulation of astrocyte transcriptional programs and implied the requirement of GSTM1 in the maintenance of neuronal activities during brain inflammation

In a depleted body, the ROS accumulate, which can cause immediate mitochondrial damage and trigger the severe, delayed symptom flares characteristic of PEM

Moreover, the significant reduction in oxidative stress provides a promising outlook on the therapeutic potential of TSS