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This shift likely represents an adaptive response to chronic excitatory activity and the metabolic burden imposed by recurrent seizures, as previously proposed in models of refractory epilepsy 16

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Drug interaction with udp-glucuronosyltransferase (Ugt) enzymes is a predictor of drug-induced liver injury

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Similarly, a higher level of C-peptide was found with a simultaneous reduction in the fasting glucose level, suggesting an insulin resistance development at the 3- and 6-month follow-up after discharge (65)