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Additionally, commonly used experimental models fail to accurately recapitulate the complex human skin aging microenvironment and multicellular interactions, particularly under the pathophysiological context of immunosenescence and structural-functional deterioration, thus impeding mechanistic-clinical alignment

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found that iron-overloaded follicular fluid could trigger ferroptosis in granulosa cells and immaturity of oocytes, thereby increasing the risk of endometriosis-related infertility [90]