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For comparison, we treated cells with tunicamycin (TUN, a widely-used ER stress inducer) which did not alter phospho-GCN2 (T899), but reduced the mobility of PERK, indicative of its increased phosphorylation (Fig

influenzae , was also found to induce MUC5AC gene expression in human lung epithelial cells and in mice trachea treated with P6 (Chen et al., 2004)

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It is hypothesised that this results in loss of activity of critical proteins and eventually hepatic cell death

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