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Normally, our bodies should make enough glutathione to protect us

Environmental toxins, chronic stress, poor sleep quality, excessive alcohol consumption, and certain medications all play a role

The oxidative damage mediated by ROS resulting in the defect in control of apoptosis or programmed cell death and delayed clearance of apoptotic cells may prolong interaction between ROS and apoptotic cell macromolecules generating neoepitopes that subsequently broad spectrum of autoantibody formation leading to the tissue damage in SLE [6,7]

This is then harvested and sent off to be used as a key ingredient in dietary supplements