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Concomitant inhibition of mdm2-p53 interaction and aurora kinases activates p53-dependent postmitotic checkpoints and synergistically induces p53-mediated mitochondrial apoptosis and reduced endoreduplication in aml

Cellular Senescence in Aging and Disease Mitigation

Once youve completed pregnancy (and breastfeeding, if applicable), your provider can help you safely resume treatment if appropriate

& Lehmann, F

Continuous ROS production results in oncogene and tumor suppressor gene changes, as well as chromosomal abnormalities by oxidative genome damage, which includes the oxidation of guanine to form 8-OhdG and 8-oxo,7,8-dihydroguanosine (8-OHG) in RNA and DNA (100)