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Further mechanistic investigations determined that the elevation of intracellular GSSG levels resulted in NF-B inhibition by facilitating glutathionylated IKK- (IKK--SSG) formation, leading to enhanced TNF hepatotoxicity

FIGURE 10 Notably, SR18 also contains a potential trypsin cleavage site (Figure 10), and the HRMS data (Supplementary Figure S7) with a peak around 685 Da confirm that trypsin can cleave SR18 between Lys17 and Ala18, leaving the remaining crucial 17 residues intact

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