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Researchers believe that its unique mechanism of action, summarized as mimicking native GIP at the GIP receptor while exhibiting bias at the GLP-1 receptor for cAMP generation over beta-arrestin recruitment, underlies its efficacy as an anti-diabetes drug [9]

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However, as with any medication that rises in popularity, misconceptions can spread

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For further details concerning the role of Se in human health (Khrle et al., 2000