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Immunohistochemical analyses of liver samples from patients with NASH demonstrated GLP-1R localization to the basolateral membranes of hepatocytes with macrovesicular steatosis, as well as in mononuclear cells infiltrating hepatic sinusoids

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However, more frequent use correlates with higher risk of complications, including electrolyte imbalances, intestinal damage, and dependency on external stimulation for bowel movements

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Some researchers believe that GIP receptor activation partially offsets the nausea caused by GLP-1 stimulation, which could explain why some patients tolerate tirzepatide better than pure GLP-1 agonists despite receiving more potent appetite suppression