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Methods Linkage Disequilibrium Score Regression (LDSC) was utilized to assess the genetic correlation between T2D and SA

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Glucagon-like peptide-1 receptor agonists (GLP-1 RAs) promote physiologic glucose-dependent insulin release

Consistently, the similar results were observed in the IMvigor210 cohort (A bladder cancer cohort treated with PDL1, Fig.7D)

Nevertheless, these findings point to complex interaction between GLP-1 and the gut, brain, immune system, and gut microbiota in control of inflammation (Figure 3 and Figure 4), offering osteoarthritis as a relevant inflammation model to explore the systemic, integrative actions of GLP-1 medicines beyond metabolic control (Figure 4)