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Most clinical monoclonal antibody drugs that rely on direct effector-cell activity are known to target antigen receptors that are buried deep inside the glycocalyx, often within 10 nm from the membrane surface 13,14 , suggesting that their effectiveness may be highly dependent upon crowding near the receptor

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Much of the discussion focuses on proposed mechanismshow TB-500 might benefit tissue repair and inflammationrather than real-world patient outcomes

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