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The field moved from single GLP-1 receptor agonism, to dual GIP/GLP-1 agonism, and then to adding a third receptor glucagon to capture an energy-expenditure lever on top of appetite and glucose control

Suppression of food and water intake by GLP-1 mainly involves interactions with various hypothalamic nuclei, particularly the arcuate and paraventricular nucleus, as well as some hindbrain structures such as the parabrachial nucleus [31]

GLP-1 receptor agonists have a sustained stimulatory effect on corticosterone release after chronic treatment

Ozempic, being widely available, provides a reliable and consistent option for most patients

Telogen effluvium does occur, and there are also associations with increased testosterone levels in the blood