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High dp-ucMGP and increased IL-6 levels together with low vitamin K levels have been identified as key factors in the inflammatory process and tissue destruction caused by COVID-19 [304, 317]

Although the role of ADE in long COVID is less well-characterized, limited evidence suggests that ADE-associated antibodies, such as anti-nucleocapsid antibodies, may contribute to disease pathogenesis primarily through immune activation (mechanism illustrated in Figure 1B) [85,86,87]

Active site residues C46 (oxidized), L48, Q81, and W136 are highlighted and shown as sticks

Further experiments confirmed the presence of vitamin E, quercetin, and cetechin, which demonstrated hepatoprotective effect, and the ROS inhibition was confirmed to be associated with antioxidant activities, thus protecting against ALI

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