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Stable at room temperature

NMDA receptors, when overactive, contribute to excitotoxicity and excessive neural firing

It upregulates genes involved in collagen synthesis (COL1A1, COL3A1, COL5A1), extracellular matrix remodeling (decorin, fibromodulin), and antioxidant defense (superoxide dismutase, glutathione peroxidase) while simultaneously suppressing genes associated with inflammation (IL-6, TNF-alpha), fibrinogen production, and tissue destruction (several MMPs)

This balanced approach can harness the benefits while minimizing uncertainties

FOXO4-DRI disrupts this interaction, freeing p53 to initiate apoptosis