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Furthermore, noise-induced tinnitus models suggest that compensatory mechanisms, potentially involving nitric oxide (NO) signaling, act to increase neuronal gain in the ventral cochlear nucleus in response to reduced input, a process that appears to involve NMDA receptor facilitation (Hockley et al., 2020)

This review focuses on the main substrates, inhibitors and reactions played by cytosolic GSTs, with an eye on their relevance for human disease

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