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Nevertheless, current GSH-activated prodrugs frequently face challenges in achieving selective activation within heterogeneous tumor microenvironments

To determine whether MRE11 mediates replication fork degradation, cells were pretreated with 50 M of the MRE11 inhibitor mirin for 30 minutes prior to CldU labeling

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Caitlin Davis from Yale University for kindly allowing us to use the OPTIR setup for additional experiments

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