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Since VIP is ineffective at stimulating gland secretion in CF, then it seems an unlikely candidate to mediate the increased muc5B concentrations (which is largely expressed in the glands and to a lesser extent in surface goblet cells [134]) that are observed during CF exacerbations [135]

This opens new avenues for PCa management, especially for patients with metabolic syndrome. The researchers highlighted that inflammation, a significant factor in cancer progression, is mitigated by GLP-1-RAs through their anti-inflammatory properties

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