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Obesity and PsA have mutually reinforcing mechanisms Excessive adipose tissue expansion induces low-grade systemic inflammation 3 Dysfunctional adipose tissue acts as an endocrine organ that secretes proinflammatory mediators relevant to PsA pathogenesis 4, 5 Obesity increases mechanical stress on joints, resulting in the microtrauma associated with PsA pathogenesis 4,5 Functional disability in PsA may reduce physical activity, favoring further weight gain 4 b Proposed mechanisms are based on current evidence and remain hypothetical

Collectively, these data might indicate that the diminished ROS buffering of GSH-deficient cells leads to an increased activation of the mTOR pathway which could drive to the MZB-like properties of Gclc -deficient FoB

Detoxification has three phases, mostly occurring in the liver

, Rosenkilde, M

This retrospective study aims to compare surgical outcomes before and following the implementation of the ASA guidelines to determine the role of GLP-1RAs in these outcomes