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It is commonly categorized as a copper peptide research compound and is often reviewed in studies involving matrix pathway behavior, collagen pathway models, extracellular matrix research, and cellular response analysis

[1] According to the researchers TGF-beta1 induction of angiogenesis requires a rapid and transient apoptotic effect mediated by VEGF/VEGFR2. AHK-Cu, by enhancing levels of the Vascular Endothelial Growth Factor, may possibly activate fibroblasts and endothelial cells

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