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Those papers which formed the preclinical rationale for dihexa's nootropic claims and for Athira's NIH grant applications had been cited as evidence that dihexa promoted synaptogenesis at rates far exceeding BDNF

GLP-1 receptor agonists slow gastric emptying, meaning food stays in your stomach longera mechanism that reduces hunger but can amplify sensitivity to certain foods

Individual response variation makes standardized protocols difficult to establish

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