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In ECs, Ca ++ flux in response to either shear stress or a PIEZO1 agonist, Yoda, leads to cellular ATP release, followed by NO synthesis downstream of the actions of the exported ATP on purinergic receptors and endothelial NO synthase activation secondary to phosphorylation of eNOS by AKT (Wang et al., 2016)

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However, the poor solubility because of strong hydrophobicity limits its further development as eIF4E PPI inhibitor