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357 Similarly, in HD and related polyQ disorders, expanded polyQ-containing proteins form insoluble aggregates that physically inhibit proteasome function, amplifying the proteotoxic burden

Jrgensen says: "We are committed as ever to diabetes

Studies show that personalized treatment matching improves adherence and reduces adverse event reporting

41 A complementary mechanism involves cardiolipin-mediated mitophagy, in which cardiolipin externalizes to the outer mitochondrial membrane and directly interacts with LC3 to trigger mitochondrial turnover

Leptin sensitivity improves as fat mass decreases