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Currently there are three phenotypes of CPTII deficiency described in literature (Joshi and Zierz, 2020): (1) the lethal neonatal form , characterized by reduced CPTII enzyme activity in multiple organs, reduced carnitine concentration, and increased concentrations of long-chain acylcarnitine and lipids in serum

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For instance, the relatively long-lived O 2 (with a half-life of 14 s and migration distance of 30 nm) generated on the mitochondrial membrane, diffuses toward the mitochondrial genome and reduces the transition metals within the genome itself