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Motoyama, K

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Similarly, PM2.5-induced increased ROS levels result in ESC cytotoxicity [278], while high osmotic stress mediated H 2 O 2 /O/OH accumulation contribuites to ESC reprogramming [279]

Establishment of comprehensive mechanistic models that incorporate mitochondrial oxidative stress, lipid metabolic reprogramming, and inflammatory amplification, to enable pathway-synergistic targeting