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Acknowledgements The authors would like to thank Henrik Kim Nielsen, Jens Heisterberg, and Karen Hauda (Novo Nordisk A/S, Denmark), and Gina MacNeil (Novo Nordisk Inc, United States) for review of, and input to, the manuscript, as well as Tina stergaard, Sisse Lakner Schmidt, and Michala Cronberg (Novo Nordisk A/S, Denmark) for help with physical stability analyses

In this study, we found that statin-induced cell death in YR1.7 and MC38 exhibited key hallmarks of ferroptosis, including downregulation of GPX4 and could be rescued by ferroptosis inhibitors (ferrostatin-1 and liproxstatin-1) for the majority of lipophilic statins

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This approach allows for the functional characterization of key genes and the exploration of their therapeutic potential

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