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Given the significant roles of NADPH in numerous pathways of reductive biosynthesis and in the maintenance of cellular redox status, in both the cytosol and the mitochondria, it is not surprising that there are multiple cytoplasmic and mitochondrial enzymes that have, as a product of their reactions, the reduced electron carrier, NADPH

Oxidative and nitrosative stress are furthermore known to promote protein S -glutathionylation [64], nitrosylation, and sulfenylation, which can regulate and affect central redox-regulated metabolic processes

According to this study (137), it has been shown that in UC, the activation of the iNOS/COX-2/5-LOX loop and increased levels of their end products, such as NO, prostaglandin E2 (PGE2), and leukotriene B 4 (LTB 4), which lead to the overproduction of free radicals and the impairment of the antioxidative system

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Antioxidant and anti-inflammatory capacities of collagen peptides from milkfish (Chanos chanos) scales[J]