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In addition to substrate binding, the NPEY motif in both IR and IGF1R is known to be involved in receptor internalization (20, 22)

The complexity of PD origins, however, has been underscored by recent discussions of brain versus gut first etiology, molecular biomarkers, involvement of S, phenotypic characteristics (such as unilateral vs bilateral, or tremor predominant vs akinetic-rigid vs postural instability, and gait disturbance), and genotyping 3,4,6,7

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Understanding your genetic predisposition to tirzepatide effectiveness allows your prescriber to personalize your treatment plan from day one, potentially improving outcomes and reducing trial-and-error adjustments

To view or add a comment, sign in New discovery from Duke University School of Medicine is rewriting the rules on blood sugar regulation