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Hallmark features of AD pathology include (I) age-dependent neurodegeneration that often manifests as gross neuroanatomical changes 12 - 14 , (II) the accumulation of pathological protein aggregates such as A plaques and neurofibrillary tangles (NFTs) 12 , (III) proliferation of glia (gliosis) and alterations in glial function that disrupt neuroimmune function and the blood brain barrier 12 , 15 , and (IV) an altered cellular landscape featuring multifaceted changes in cell signaling and metabolism, excessive oxidative stress, dysfunctional autophagy, impaired cell transport, and synaptic dysfunction 16 - 19

The fact that the comparisons were statistically significant does, in fact, demonstrate that the sample size was adequate.The most common objection to the study of Schultz et al

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Inhibition and potentiation of LHRH by closely related analogues