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Excessive Ca 2+ production is another way of nanoparticle induced toxicity, as it induces apoptosis via releasing cytochrome c [335]

citrulline malate, both forms share the same primary pathways: they participate in the urea cycle, helping to clear ammonia from the bloodstream, which delays fatigue, and they stimulate nitric oxide production to widen blood vessels and improve nutrient delivery to working muscles

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( : The New England Journal of Medicine) [ ]