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In particular, for patients who do not respond to conventional therapies, the regulatory mechanisms of the BGA may represent important intervention targets (Figure 1)

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Through its action, this peptide is deactivated as it is converted to the oxidised form ( Reference Dringen, Gutterer and Hirrlinger 17 )

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It is possible that improvement in nerve size following GLP-1 RA treatment may be due to augmentation of pump activity and normalisation of the sodium electrochemical gradient, as suggested by previous human and animal studies [7]