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Through Kla-mediated epigenetic rewiring, lactate actively suppresses antitumor immunity while potentiating oncogenesis (60, 61)

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In addition to the acquirement of drug-resistant mutation, the development of cancer stem cells also causes CML persistence and relapse from TKI therapy 12,13,14

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Finally, in vitro studies have shown the potential of NAC (1nM) to disassemble fibrils, protofibrils, oligomers and dimers of A molecules by blocking continuity in their oligomerization, a phenomenon mediated by the formation of hydrogen bridges between the A molecule and NAC, and by the stabilization of the neprilysin protease which is part of the catabolic equilibrium of the A peptide [70,71,72]