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This distinction underscores the importance of viewing GLP-1RAs as systemic immunometabolic modulators with secondary effects on thyroid autoimmunity, rather than as agents with direct thyroidal targets

Proc Natl Acad Sci USA 93(26):15346-15351, 1996

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That extra ATP sets in motion a chain of events that increase mitochondrial oxidative stress, and ultimately destroys the electrical membrane potential that mitochondria need for normal respiration

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