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Given AKT1s established role in insulin signaling, mitochondrial function, and energy metabolism, its upregulation may explain key pathological features of lipedema, including impaired oxidative phosphorylation, chronic inflammation, and altered adipocyte metabolism

Its name comes from early findings that it increased delta-wave activity on EEG, which corresponds to deep, restorative sleep

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This is at least in part due to its adverse effects on the body's stores of vitamins and enzymes and the oxidative defense system