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While the chemically induced models of PD, MPTP and 6-OHDA, provide valuable insight into the oxidative stress-related mechanisms of dopaminergic neuron loss in vivo , they do not fully recapitulate PD neuropathology with -syn aggregation (Meredith and Rademacher, 2011

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Critically, AOD-9604 does NOT stimulate growth hormone receptor (GHR) signaling in the classical sense

Pharmacology [edit] Metabolism and half-life [edit] Tesofensine has a long half-life of about 9 days (220 h) [4] "and is mainly metabolized by cytochrome P4503A4 (CYP3A4) to its desalkyl metabolite M1" NS2360

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