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10.25259/IJMR_497_2025 Indian J Med Res

DNMT1-mediated methylation of BEX1 regulates stemness and tumorigenicity in liver cancer

From that biology, researchers draw a theoretical concern: chronically potentiating a proliferative, pro-angiogenic pathway is a plausible mechanism by which an agent could, in principle, support growth of existing malignancy

To advance science, we must provide these tools to scientists, but their use outside of these controlled environments is inappropriate and, in the case of athletes, a violation of the rules

This vulnerability has been targeted in therapies with GPX4 inhibitors or iron modifiers in order to increase lipid peroxidation and preferentially kill cancer cells