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Further, these data suggest that at acute time points following Gclc genetic deletion, compensation by NRF1 could be responsible for the dispensable role of NRF2 in GSH-depleted conditions

by Crystal A

A.GriffinJ

These changes are not always tied to a single lab value

In research models, increased expression of extracellular matrix proteins has been observed, supporting its use in studies of wound healing and skin regeneration