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[11] [4] Preclinical studies have suggested that combining amylin receptor agonism with GLP-1based therapies produces greater reductions in food intake and body weight than either approach in isolation, and early clinical data from the CagriSema programme (cagrilintide combined with semaglutide) has provided some supporting evidence in humans

Leptin exhibits clear pro-carcinogenic effects by promoting systemic inflammatory responses and cellular proliferation, while inhibiting apoptosis and immune surveillance functions, whereas adiponectin exerts the opposite effects [11]

Curr Neuropharmacol (2021) 19(5):591609