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Rather than targeting IGF-IR/InsR themselves, an alternative strategy would be inhibition of the master regulators of RTKs or downstream signaling hubs such as IRS1/2 [205,206,207,208]

M., & Kovalev, G

We propose that the dosage and duration of GSH administration in clinical treatment be carefully evaluated and standardized to prevent overuse and mitigate the risk of chemotherapy resistance

We also found that GSSG inhibits IDE while GSH increased the breakdown of partially-degraded insulin