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Experimental models demonstrate that restoring glutathione normalizes mitochondrial redox status and improves endothelial and cardiovascular function, supporting a causal role for mitochondrial glutathione depletion in cardiovascular aging (17-18)

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Compound specification The reference identifiers researchers will need for procurement records and assay documentation: The N-terminal acetyl group and C-terminal amide protect the sequence against exopeptidase cleavage, preventing enzymatic degradation during storage and handling

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