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Read in context, it is a secondary, exploratory outcome from a Phase 2 trial whose primary endpoint, change in cerebral glucose metabolism, was negative, and whose cognitive signal was modest

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These results suggest a potential regulatory relationship between thymosin beta 4, microRNA 146a expression, and downstream inflammatory pathway modulation

[4] A related and important point of nuance is that copper peptides can be antioxidant or pro-oxidant depending on how the copper is coordinated