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Interestingly, deoxycholic acid-chitosan coated liposomes combined with in situ colonic gel enhances renal fibrosis therapy of emodin (Xu et al., 2022)

This nutrient acts as the gate-keeper that allows fatty acids to pass into the mitochondrial furnace effectively (7, 8, 9)

Furthermore, mitochondrial dysfunction is closely implicated in AKI pathogenesis through the activation of inflammatory signaling cascades, which may lead to PTC apoptosis and necrosis (Sun et al., 2019)

most commonly, patients reported taking metformin (91%) and insulin glargine or an insulin glargine biosimilar (50%)

Regular monitoring ensures continued progress and safety